Shortening URLs (web links) becomes necessary when you need to share a long URL somewhere. As long links look ugly and take a lot of space, we have to use a web service that produces short forms of the URLs we provide. These services keeps the original URLs in their database and redirects short URLs to the original ones. Thus public URL shortener services are a lot helpful to shorten long URLs.
If you are someone who have to shorten URLs from time to time, visiting as URL shortener service and manually converting the URLs cost you extra time. Having a shortening system integrated into your browser that will let you shorten your current page URL with one click saves a lot of time. Previously, I have mentioned about an extension that lets you shorten URLs right from Chrome context menu. Here I will show two Firefox add-ons that integrates similar and more options.
Shortly URL Shortener
With Shorty, a simple Firefox add-on, you can just right-click on your page of which you want to shorten URL to get the option to shorten. Shortly will instantly produce a shortened URL with a notification that will be copied to your clipboard. You will be able paste the link anywhere you want.
Alternatively you can click on Shortly button on Firefox toolbar. It uses the URL shortener service bit.ly. However it doesn’t have option to use any other service of your choice.
Shortly Features
- Right-click entry and Toolbar button.
- Copy URL to clipboard.
- Customize keyboard shortcut.
- Show shortened URL near address bar.
Cutyfox URL Shortener
Unlike Shortly, Cutyfox comes with extra option to choose the shortener service you want.
For bit.ly, you can also use sign in. Like Shortly, it also adds a shortcut button to toolbar.
Cutyfox Features
- Right-click menu and toolbar button.
- Instantly copy to clipboard.
- Option to change Keyboard shortcut.
- Bit-ly authentication.
- Top Services bit.ly – j.mp, goo.gl, mcaf.ee and is.gd.
- Customize display options.
If you are unsure which add-on to use, I suggest to choose the first one, I mean Shortly, which is very simple and ready. Shortening URLs right from your Firefox browser was never been easy.
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Hello! I'm Dr Trites with Doctors Optimum, and today I'm going to fairly share
Vitamin D. So what is Vitamin D? It's actually really not a supplement at all.
It's a fat-soluble hormone, and it is contained in very few foods but is added to numerous
others.
It is a dietary supplement, it can be in a fluid form and it may
be in powder form, and our research indicates that it is actually better in a fluid
form. More to the point, it's better in a liquid form that also posseses
an added fat. Now in the event that you flip the label of one's Vitamin D, it might be with Vitamin E, which is a liquid fat-soluble vitamin, with a mixed D- alpha-tocopherol, may
even maintain fish oil because that's fat, but ours comes in what's called MCT,
which is a medium-chain triglyceride.
It's essentially a form of fat, and medium-chain triglycerides can be found in many forms:
it may be olive oil, or maybe it's coconut oil, or it could be sesame seed oil, or maybe it's any one
of those particular forms, and that's called the medium-chain triglycerides.
Those are what's called'good fats ', like what's present in an avocado.
Now, some people would say, "Well, I went outside today, so I acquired lots of Vitamin D." And that's true:
the human body does trigger an answer once the sunlight
hits skin, it will, what's called'synthesize'the C hormone into a..
well, there's a difference between D-3 and D-2, and it synthesizes and changes it.
So bear in mind, Vitamin D was initially found and thought to be a supplement, but really it
is a hormone, and it's calciferol or cholecalciferol,
and there's ergocalciferol, which is the D-2 form.
D-2 is normally what you will find in the pharmaceutical
brands, and that is what is allowed to be pharmaceutical.
D-3 is more on the supplemental side. It's deemed to become more biologically active.
It has receptor sites or more receptors compared to D-2 does.
And in addition, D-2 may be converted, that's
what they are able to enter the pharmaceutical side and hope
that it actually raises the D count. Why take Vitamin D?
There are a large amount of problems that Vitamin D has been shown to help out, and
we also do a blood test if you have a question at all, to find
out, ”Is Vitamin D in your particular body adequate?
Do you need a supplement with Vitamin D, and once you supplement with Vitamin D, achieved
it raise your values as to the is known as normal or not?"
So just why in case you even take our Vitamin D? Well, number 1, it is in a liquid form. In the event that you remember, that liquid form is a little bit more viable than the usual powder form. Our particular liquid could be taken alone, or it could be added to any liquid.
We formulate it because we wanted to ensure it's the best Vitamin D and that it has fat with it called the MCT - the medium-chain triglyceride. And we've plenty of studies that report, including many documentations within our personal office, to show it's a decent supplement only for muscular pain, low back pain, diabetes, auto-immune conditions - it must be there.
Obviously, we use the biologically active D-3 form. We didn't want to go with D-2, and it's not in a pill form, so you can bring it in at any age. You can just drop it or add it to anything. And needless to say, our products at Doctors Optimum are without any sugar, salt, starch, yeast, wheat, gluten, soy, milk, egg, shellfish and preservatives. Again, I'm Dr Trites at Doctors Optimum, be well.
Dr Killin mentioned our research can be highly linked to the FDA our research is under investigator-initiated IND because clearly
the goal of our research is to stop treat mitigate or cure relating to the prevention and treatment of infectious diseases at the extremes of age or
in immunocompromise so we're greatly under regulation from the FDA
that has changed dramatically just how we move ahead so
in 2006 it had been all so simple I was very very fortunate to
have this NIH grant-funded and the concept here was simple elderly people do not respond well to the flu vaccine they require some assistance probiotics may possess
some immunomodulatory properties nevertheless the indisputable fact that probiotics would actually benefit a
flu shot was a little far-fetched however how about that new cosily administered flu vaccine that clearly has a niche site of action in the mucosa where the probiotics have a niche site of action is it feasible we could increase the
immune response of the flu vaccine for elderly subjects.
So we were very excited to talk to the FDA and they said reasonably so their argument
was you've got showing that the product is safe and also the way the heck have you any idea it's safe in older people have you truly seriously considered
that and the answer was not likely around we must just what exactly the FDA asked
us to do and I'm likely to report a little data run today is
first do some thing just super simple can elderly people safely tolerate
a probiotic over a brief time period and when they will then what I'm going to do is move ahead to see whether
or not administering the probiotic with the flu shot helps honestly didn't really expect
it would but then and unfortunately at the conclusion of the flu
season because the flu mist is not approved for individuals over
the ripe later years of 49 ouch underneath line is we then wanted to maneuver ahead with this particular
to gauge safety and preliminary immunogenicity and to potentially study the mechanism of action of the probiotics in this particular circumstance then and this really is where we're
right now we are able to return back and communicate with the FDA and see what they think
about proceeding with the original study.
In order that open label study in elderly people we found 15 of these aged 65 to 80 it really is amazing how many individuals truly
think they're healthy yet have a big quantity of conditions we've people
who came in we asked if you'd they'd diabetes understand what meds are you taking insulin well don't you have diabetes no the insulin cures
the diabetes I don't have diabetes anymore so you've clearly got to essentially do these studies properly to be able to be sure you have healthy
people but I was also amazed to find individuals 80 years old who aren't on a single medication it happens
we'd these subjects consume lactobacillus gg a reasonably high dose twenty-eight days and
then we followed them for another 28 days we followed their safety
on a regular basis prompting them if they'd Domino complaints or any other issues
we then called them up we had study visits and in this circumstance along side Dr Fraser, we were able to do a few of the microbiome and omics studies and with Dr.
Gloria, we could actually do a whole blood cell transcriptome studies our safety data are honestly
moderately boring.
But that's good I can tell you among the things we're
excellent at is we enjoy to follow up these subjects and we have great track
records this type of subjects who participate in these studies often love these
studies they love filling out their daily Diaries oh my goodness once we stopped asking them for the daily Diaries many were crushed what were they going to
accomplish but clearly when we're asking people all the time if they've
any negative effects we are going to have them and yes in 15 people 47 unwanted effects sound such as for instance a
lot but they certainly were all mild meaning they did nothing they didn't take
any meds they didn't visit the dock nothing else was going on and
we did have just one single severe unrelated adverse event in a person who passed
a kidney stone but apart from that totally trivial negative effects what this slide shows in collaboration with dr.
Gloria is that when we actually look the LGG in the stool that we get
from these subjects and this is at day 28 and they were moderately compliant there clearly was only one subject
who had been not compliant at all there's lots of variability in the of counts of LGG using PCR
which does grab dead and alive organisms but in addition should you choose routine culture you'll also see there's lots of variabilities does this matter I don't know.
But with this really is among the things that we're delving into at this time and hasn't been well addressed in the literature so the recovery of
LGG is variable by subject and it is also not the same by
method because the techniques are doing different things this work done with
dr. Fraser at Maryland basically may be the 16s
ribosomal RNA information so it's not too whole pair of gene information it's
more limited set and what this shows is these are the three days baseline day 28
and day 56 and that's repeated for each of the subjects and I think
you can see overall this looks incredibly boring like there's not much going on at all and in the event that you give attention to the phylogenetic diversity over here
which can be which range from twenty to forty per cent you'll see it mostly bounces around and stays flat therefore
the 16s level give people LGG for 28 days very little happens he is mildly
interesting he was prescribed Cipro and his diversity dropped dramatically about six
weeks into therapy.
So that was probably the most exciting thing that we found so far
as which was concerned this again is really a representation of that 16s data however what Dr Fraser did
was the whole gene data and is it possible to begin to see the pans general a similar but the
info are very different again telling us we got to be awfully careful what
we're considering and we really should understand the questions that individuals are asking the one thing which was very
interesting here's we're picking right up all this yellow
stuff will be the archaea not in everybody in seven of
the individuals but it doesn't seem like it's changing in virtually any particular way what're these archaea doing again something pretty
interesting and then this really is really cool what
dr. Fraiser did was look at the LGG itself can there be anything going
up with LGG at baseline nope on day 28 every one of these
yellow indicate that the probiotic is clearly making copies or there's in vitro expression of the genes
associated with LGG the sole person that people didn't find any expression in was somebody who wasn't taking it.
That is a relief and on day 56 gone so three other ways of looking
at the information telling us different things but what this probably
tells us the thought of lobbying in LGG changing what's going
on in the gut in terms of the bugs that are they're not likely going to happen however it doesn't mean that there aren't subtle effects and
these data from dr. Gloria's lab are also very interesting they they're PACs
gene RNA from the host so we're now taking a look at the host immune response remember
they certainly were healthy elderly people who just took LGG there clearly was nothing
going on we weren't treating anything more nevertheless the was a differential expression at day 28 versus
day zero and the gene that struck out the absolute most in this
circumstance happened to be a gene that was downregulated related to the regulation of IgE the effects with this again were investigating further.